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In a trial conducted on a small number of subjects with Alzheimers disease, treatment with liraglutide was able to prevent the progressive decline in cerebral glucose uptake that characterizes patients with the disease [24]

By contrast, M2-like TAMs create a ferroptosis-resistant niche through high ferroportin expression, heme degradation pathways (HMOX1), the generation of antioxidant glutathione, and the secretion of IL-10 and TGF- (72)

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