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As regards the pathogenic mechanisms, the occurrence of an oxidant stress (resulting from an impaired homocysteine metabolism, which modifies the redox status of extra- and intracellular thiols and thus alters the intracellular redox potential) might play a central role in moderate hyperhomocysteinemia-mediated vascular disorders

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Apigenin blocked lipopolysaccharide (LPS)-induced cytotoxicity, apoptosis, and inflammatory cytokine secretion by suppression of the TLR4/MyD88/NF-B pathway [142]

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"Notice of interim decisions to amend (or not amend) the current Poisons Standard