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Glucagonlike peptide-1 7-36: a physiological incretin in man

However, more recent studies assessing the role of ferrous iron in the pathophysiology first in vitro [74, 75] and then in vivo [76, 77] provided evidence that after a toxic dose of APAP ferrous iron derived from lysosomes is taken up into mitochondria through a Ca 2+ uniporter and facilitates the MPTP opening and cell death

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Our results showed that UAc increase as dietary L-carnitine, Lys and Met intake increases

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