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Specifically, we provide evidence that CD38 could be considered as an immunometabolic checkpoint, joining the therapeutic promise of other immune checkpoints previously proposed to modify AD 54 and other forms of dementia 40

But in any case, its only a temporary shift

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A similar strategy was used by DallAcqua and colleagues to generate human IgG1 variants with increased affinity for both mouse and human FcRn [100]

Its a matter of skippering between the two