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h , Bar graph summarizing the effect of NMDA-induced inward current after control (aCSF, n = 5 neurons), GLP-1 (50 M, n = 5 neurons), MK-801 (50 M, n = 5 neurons) or GLP-1MK-801 (50 M, n = 5 neurons)

J Neurochem (2003) 87(5):113744

In 2024 the journal Diabetes named it as a classic paper

(54) found that administering GIPR antagonist to healthy humans reduced postprandial insulin secretion and increased glucose excursions more than administering GLP-1R antagonist (GLP-1RA), indicating that endogenous GIP may play a more significant role in stimulating insulin secretion and reducing postprandial plasma glucose excursions compared to endogenous GLP-1

doi:10.1172/JCI112855