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Critical constituents of PPO comprise two conserved Cu-binding domains and chloroplast and thylakoid transit peptides at the N-terminal [186]

Mutations in genes affecting the production of this enzyme can have a significant impact on the function of the MTR enzyme and thus recycling of homocysteine back into methionine (13)

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The significant reduction in Bacteroides abundance observed during OA onset supports the protective role of Bacteroides in OA pathogenesis, potentially mediated through intestinal homeostasis maintenance (Kurz et al., 2025)

The generated Fe + ions form a stable ferri-Tiron complex through specific coordination chemistry